This is real... it's based on verifiable science...and it flat-out WORKS.

And don't worry if you're not a "science" type... I'm going to break the whole "craving killer" process down into straightforward language so you'll see exactly what's going on the whole way through.

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Step 1 - You have bugs (i.e. bacteria) in your gut....some "good"... some "bad."

• Gut bacteria can secrete neurotransmitters (brain chemicals) and hormones that affect your behavior, mood and thoughts... they can even make you feel hungry when you're not. [1,2]
• Gut bacteria can become very specialized in what foods it prefers. [3,4,5,6] For example, SUGAR.

Step 2 - When you go on a diet and/or don't eat sugar, those specialized bacteria start to die and emit "death screams" in the form of those neurotransmitters and hormones [7,8] in an attempt to get you to eat sugar to feed themselves and not die.

Step 3 - If you give in, you start the cycle all over again. You feed the "bad" bacteria and they stay alive until you try again...setting up a constant cycle of cravings and failure.

Step 4 - With this "3-Day Craving Killer Protocol," we KILL those "bad" bacteria using targeted supplementation before they even have a chance to scream.

Step 5 - Then we repopulate with "good" bacteria (and one other critical component).

Problem solved. No more cravings. Just with a few specialized, easily-available, inexpensive supplements.

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Now, naturally this is a simplification of the process...however it pretty well sums it up.

When I first created the 3-Day Craving Killer Protocol, I thought to myself "There's no way it can be this simple."

Until I used it on myself...and it WAS that simple.

The results were clear and the science backs it up. This works and it works FAST.

Learn more about The 3-Day Craving Killer Protocol and how it wil work to end your cravings FAST here...

Nick Nilsson
The Mad Scientist of Muscle

REFERENCES

[1.] Fetissov S, et al. (2008). Autoantibodies against appetite-regulating peptide hormones and neuropeptides: Putative modulation by gut microflora. Nutrition. April 2008 Volume 24, Issue 4, Pages 348–359

[2.] Barrett E, et al. (2014). γ-Aminobutyric acid production by culturable bacteria from the human intestine. J Appl Microbiol. 2014 May;116(5):1384-6.

[3.] Gonzalez-Rodriguez I, Ruiz L, Gueimonde M, Margolles A. (2013). Factors involved in the colonization and survival of bifidobacteria in the gastrointestinal tract, FEMS Microbiology Letters, Volume 340, Issue 1, 1 March 2013, Pages 1–10

[4.] Wu GD, Chen J, Hoffmann C, Bittinger K, et al. (2011. Linking long-term dietary patterns with gut microbial enterotypes, Science  07 Oct 2011: Vol. 334, Issue 6052, pp. 105-108

[5.] Walter J, Ley R, (2011). The human gut microbiome: ecology and recent evolutionary changes, Annual Review of Microbiology, Vol. 65:411-429 (Volume publication date October 2011) 

[6.] Rezzi S, Ramadan Z, Martin FP, Fay LB, (2007). Human metabolic phenotypes link directly to specific dietary preferences in healthy individuals, J. Proteome Res., 2007, 6 (11), pp 4469–4477

[7.] Chiu I, et al. (2013). Bacteria activate sensory neurons that modulate pain and inflammation. Nature, September 2013, 501, 52–57

[8.] Chen C, Pande K, French SD, Tuch BB , et al. (2011). An iron homeostasis regulatory circuit with reciprocal roles in Candida albicans commensalism and pathogenesis. Cell Host Microbe 10 : 118–35.

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